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The results indicated that Lactobacillus might contribute to pathogenesis of OA by involving iron metabolism and ferroptosis

However, the identity of specific amino acids that are sensed by mTORC1 are presently unknown (Jewell et al

Oxidized LDL specifically promotes the initiation of monocyte invasion during transendothelial migration with upregulated PECAM-1 and downregulated VE-cadherin on endothelial junctions

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